High resolution integrative analysis reveals widespread genetic and epigenetic changes after chronicin-vitroacid and bile exposure in barrett's epithelium cells

Author:

Bajpai Manisha1,Kessel Rachel2,Bhagat Tushar2,Nischal Sangeeta2,Yu Yiting2,Verma Amit2,Das Kiron M.1

Affiliation:

1. Division of Gastroenterology and Hepatology, Department of Medicine, RUTGERS Robert Wood Johnson Medical School; Rutgers Cancer Institute of New Jersey; New Brunswick NJ

2. Department of Medicine (Oncology); Albert Einstein College of Medicine; Bronx NY

Publisher

Wiley

Subject

Cancer Research,Genetics

Reference60 articles.

1. Role of epigenetic alterations in the pathogenesis of Barrett's esophagus and esophageal adenocarcinoma;Agarwal;Int J Clin Exp Pathol,2012

2. Widespread hypomethylation occurs early and synergizes with gene amplification during esophageal carcinogenesis;Alvarez;PLoS Genet,2011

3. Repeated exposure to acid and bile selectively induces colonic phenotype expression in a heterogeneous Barrett's epithelial cell line;Bajpai;Lab Invest,2008

4. Prolonged exposure to acid and bile induces chromosome abnormalities that precede malignant transformation of benign Barrett's epithelium;Bajpai;Mol Cytogenet,2012

5. Determination of the frequency of loss of heterozygosity in esophageal adenocarcinoma by cell sorting, whole genome amplification and microsatellite polymorphisms;Barrett;Oncogene,1996a

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