Absence of<scp>IκBβ</scp>/<scp>NFκB</scp>signaling does not attenuate acetaminophen‐induced hepatic injury-Reference-Cited by-同舟云学术

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Absence ofIκBβ/NFκBsignaling does not attenuate acetaminophen‐induced hepatic injury

Published:2022-11-25 Issue: Volume: Page:
ISSN:1932-8486
Container-title:The Anatomical Record
language:en
Short-container-title:The Anatomical Record

Author:

Solar Mack¹,Grayck Maya R.¹,McCarthy William C.¹,Zheng Lijun¹,Lacayo Oscar A.¹,Sherlock Laura G.¹,Zhou Ruby¹,Orlicky David J.²,Wright Clyde J.¹^ORCID

Affiliation:

1. Section of Neonatology, Department of Pediatrics University of Colorado School of Medicine Aurora Colorado USA

2. Department of Pathology University of Colorado Anschutz School of Medicine Aurora Colorado USA

Funder

Eunice Kennedy Shriver National Institute of Child Health and Human Development

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Ecology, Evolution, Behavior and Systematics,Histology,Biotechnology,Anatomy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ar.25126

Reference73 articles.

1. Differential Expression of Mouse Hepatic Transporter Genes in Response to Acetaminophen and Carbon Tetrachloride

2. Nuclear Factor-κB Activation in Neonatal Mouse Lung Protects against Lipopolysaccharide-induced Inflammation

3. Mitochondria to nucleus stress signaling

4. Mitochondria-to-nucleus stress signaling in mammalian cells: Nature of nuclear gene targets, transcription regulation, and induced resistance to apoptosis

5. Dioxin-mediated tumor progression through activation of mitochondria-to-nucleus stress signaling

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