Catalpol ameliorates advanced glycation end product‐induced dysfunction of glomerular endothelial cells via regulating nitric oxide synthesis by inducible nitric oxide synthase and endothelial nitric oxide synthase-Reference-Cited by-同舟云学术

Catalpol ameliorates advanced glycation end product‐induced dysfunction of glomerular endothelial cells via regulating nitric oxide synthesis by inducible nitric oxide synthase and endothelial nitric oxide synthase

Published:2019-03-12 Issue:9 Volume:71 Page:1268-1283
ISSN:1521-6543
Container-title:IUBMB Life
language:en
Short-container-title:IUBMB Life

Author:

Sun Weixiang¹²³,Gao Yuyan¹²,Ding Yushi¹²,Cao Ying³,Chen Jing¹²,Lv Gaohong¹²,Lu Jinfu¹²,Yu Bin¹²,Peng Meilin¹,Xu Huiqin¹²³^ORCID,Sun Yun³

Affiliation:

1. Department of Pharmacology, School of PharmacyNanjing University of Chinese Medicine Nanjing People's Republic of China

2. Jiangsu Key Laboratory for Pharmacology and Safety Evaluation of Chinese Materia Medica, Nanjing University of Chinese Medicine Nanjing People's Republic of China

3. Department of Pharmacology, School of PharmacyHanlin College, Nanjing University of Chinese Medicine Taizhou People's Republic of China

Funder

Priority Academic Program Development of Jiangsu Higher Education Institutions

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/iub.2032

Reference67 articles.

1. Endothelial dysfunction and pathogenesis of diabetic angiopathy