Disrupted cellular calcium homeostasis is responsible for Aβ‐induced learning and memory damage and lifespan shortening in a model of Aβ transgenic fly-Reference-Cited by-同舟云学术

Disrupted cellular calcium homeostasis is responsible for Aβ‐induced learning and memory damage and lifespan shortening in a model of Aβ transgenic fly

Published:2022-05-09 Issue:8 Volume:74 Page:754-762
ISSN:1521-6543
Container-title:IUBMB Life
language:en
Short-container-title:IUBMB Life

Author:

Cheng Kuan‐Chung¹²,Huang Chih‐Yuan³⁴,Hsieh Tsung‐Chi¹²⁵,Chiang Hsueh‐Cheng¹²^ORCID

Affiliation:

1. Department of Pharmacology, College of Medicine National Cheng‐Kung University Tainan Taiwan

2. Institute of Basic Medical Sciences, College of Medicine National Cheng‐Kung University Tainan Taiwan

3. Division of Nephrology, Department of Internal Medicine Ditmanson Medical Foundation Chia‐Yi Christian Hospital Chiayi Taiwan

4. Department of Sport Management, College of Recreation and Health Management Chia Nan University of Pharmacy and Science Tainan Taiwan

5. Brain Research Center National Tsing Hua University Hsinchu City Taiwan

Funder

Ministry of Science and Technology, Taiwan

Ministry of Education

Ditmanson Medical Foundation Chia-Yi Christian Hospital

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/iub.2621

Reference35 articles.

1. Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein production

2. The Swedish mutation causes early-onset Alzheimer's disease by β-secretase cleavage within the secretory pathway

3. Translating cell biology into therapeutic advances in Alzheimer's disease

4. Signaling pathway cross talk in Alzheimer’s disease

5. AlzPathway: a comprehensive map of signaling pathways of Alzheimer’s disease

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Sevoflurane exposure causes neuronal apoptosis and cognitive dysfunction by inducing ER stress via activation of the inositol 1, 4, 5-trisphosphate receptor;Frontiers in Aging Neuroscience;2022-10-20