A Kinase-Independent Function of c-Src Mediates p130Cas Phosphorylation at the Serine-639 Site in Pressure Overloaded Myocardium-Reference-Cited by-同舟云学术

A Kinase-Independent Function of c-Src Mediates p130Cas Phosphorylation at the Serine-639 Site in Pressure Overloaded Myocardium

Published:2015-10-08 Issue:12 Volume:116 Page:2793-2803
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Palanisamy Arun P.¹,Suryakumar Geetha¹,Panneerselvam Kavin¹,Willey Christopher D.¹,Kuppuswamy Dhandapani¹

Affiliation:

1. Division of Cardiology, Department of Medicine; Gazes Cardiac Research Institute, Medical University of South Carolina; Charleston South Carolina 29425-2221

Funder

Program Project

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference27 articles.

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3. Site-specific microtubule-associated protein 4 dephosphorylation causes microtubule network densification in pressure overload cardiac hypertrophy;Chinnakkannu;J Biol Chem,2010

4. 1973. Mechanisms for the abnormal energetics of pressure-induced hypertrophy of cat myocardium;Cooper;Circ Res

5. Cell adhesion regulates the interaction between the docking protein p130(Cas) and the 14-3-3 proteins;Garcia-Guzman;J Biol Chem,1999

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