PM2 .5‐induced pulmonary inflammation via activating of the NLRP3 /caspase‐1 signaling pathway-Reference-Cited by-同舟云学术

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PM2 .5‐induced pulmonary inflammation via activating of the NLRP3 /caspase‐1 signaling pathway

Published:2020-09-30 Issue:3 Volume:36 Page:298-307
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Jia Hui¹,Liu Yang¹,Guo Dan¹,He Wei¹,Zhao Long¹,Xia Shuyue¹^ORCID

Affiliation:

1. Department of Respiratory and Critical Care Medicine Central Hospital Affiliated to Shenyang Medical College Shenyang China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.23035

Reference35 articles.

1. Breathing-rate adjusted population exposure to ozone and its oxidation products in 333 cities in China

2. Diesel particulate matter2.5 promotes epithelial-mesenchymal transition of human retinal pigment epithelial cells via generation of reactive oxygen species

3. Long‐term effect of personal PM(2.5) exposure on lung function: a panel study in China;Zhou Y;J Hazard Mater,2020

4. Airborne Bacteria Enriched PM2.5 Enhances the Inflammation in an Allergic Adolescent Mouse Model Induced by Ovalbumin

5. Preventive effect of ursolic acid derivative on particulate matter 2.5‐induced chronic obstructive pulmonary disease involves suppression of lung inflammation

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3. Lung microbiota participated in fibrous microplastics (MPs) aggravating OVA-induced asthma disease in mice;Food and Chemical Toxicology;2024-08

4. Disease types and pathogenic mechanisms induced by PM2.5 in five human systems: An analysis using omics and human disease databases;Environment International;2024-08

5. Impact of Respiratory Dust on Health: A Comparison Based on the Toxicity of PM2.5, Silica, and Nanosilica;International Journal of Molecular Sciences;2024-07-12

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