The transmembrane protein LRIG1 triggers melanocytic tumor development following chemically induced skin carcinogenesis-Reference-Cited by-同舟云学术

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The transmembrane protein LRIG1 triggers melanocytic tumor development following chemically induced skin carcinogenesis

Published:2021-03-31 Issue:8 Volume:15 Page:2140-2155
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Hoesl Christine¹^ORCID,Fröhlich Thomas²,Posch Christian³⁴,Kneitz Hermann⁵,Goebeler Matthias⁵,Schneider Marlon R.¹,Dahlhoff Maik⁶^ORCID

Affiliation:

1. Institute of Molecular Animal Breeding and Biotechnology Gene Center LMU München Germany

2. Laboratory for Functional Genome Analysis (LAFUGA) Gene Center LMU München Germany

3. Klinik und Poliklinik für Dermatologie und Allergologie Klinikum rechts der Isar – TU München Germany

4. Faculty of Medicine Sigmund Freud Universität Wien Austria

5. Klinik und Poliklinik für Dermatologie, Venerologie und Allergologie Universitätsklinikum Würzburg Germany

6. Institute of In vivo and In vitro Models University of Veterinary Medicine Vienna Austria

Funder

Else Kröner-Fresenius-Stiftung

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.12945

Reference47 articles.

1. The Global Burden of Skin Disease in 2010: An Analysis of the Prevalence and Impact of Skin Conditions

2. ERBB2 Is Essential for the Growth of Chemically Induced Skin Tumors in Mice

3. ERBB3 is required for tumor promotion in a mouse model of skin carcinogenesis

4. ErbB receptors mediate both migratory and proliferative activities in human melanocytes and melanoma cells

5. The receptor tyrosine kinase ERBB4 is expressed in skin keratinocytes and influences epidermal proliferation

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2. MicroRNA-218-5p accelerates malignant behaviors of breast cancer through LRIG1;Clinics;2023-01

3. ERBB3 binding protein 1 promotes the progression of malignant melanoma through activation of the Wnt/ β-catenin signaling pathway;Cancer Cell International;2022-01-29

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