EBP2, a novel NPM‐ALK‐interacting protein in the nucleolus, contributes to the proliferation of ALCL cells by regulating tumor suppressor p53-Reference-Cited by-同舟云学术

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EBP2, a novel NPM‐ALK‐interacting protein in the nucleolus, contributes to the proliferation of ALCL cells by regulating tumor suppressor p53

Published:2020-11-19 Issue:1 Volume:15 Page:167-194
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Uchihara Yuki¹^ORCID,Tago Kenji²^ORCID,Tamura Hiroomi¹^ORCID,Funakoshi‐Tago Megumi¹^ORCID

Affiliation:

1. Division of Hygienic Chemistry Faculty of Pharmacy Keio University Tokyo Japan

2. Division of Structural Biochemistry Department of Biochemistry Jichi Medical University Shimotsuke‐shi Japan

Funder

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.12822

Reference59 articles.

1. Anaplastic lymphoma kinase: signalling in development and disease

2. ALKoma: A Cancer Subtype with a Shared Target

3. Mechanistic insight into ALK receptor tyrosine kinase in human cancer biology

4. Anaplastic Large Cell Lymphoma

5. TPM3-ALK and TPM4-ALK Oncogenes in Inflammatory Myofibroblastic Tumors

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1. Uncovering the Multifaceted Roles of DDX21: Bridging Biological Insights and Medical Applications;Journal of Bio-X Research;2024-01

2. Proteomics analysis identifies the ribosome associated coiled‐coil domain‐containing protein‐124 as a novel interaction partner of nucleophosmin‐1;Biology of the Cell;2023-12-10

3. The Leukemic Phase of ALK-Negative Anaplastic Large Cell Lymphoma Is Associated with CD7 Positivity, Complex Karyotype, TP53 Deletion, and a Poor Prognosis;Cancers;2021-12-16

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