UDP‐glucose dehydrogenase expression is upregulated following EMT and differentially affects intracellular glycerophosphocholine and acetylaspartate levels in breast mesenchymal cell lines-Reference-Cited by-同舟云学术

UDP‐glucose dehydrogenase expression is upregulated following EMT and differentially affects intracellular glycerophosphocholine and acetylaspartate levels in breast mesenchymal cell lines

Published:2022-02-03 Issue: Volume: Page:
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Molecular Oncology

Author:

Wang Qiong¹,Karvelsson Sigurdur Trausti¹,Johannsson Freyr¹,Vilhjalmsson Arnar Ingi¹,Hagen Lars²³⁴,Miranda Fonseca Davi²³⁴,Sharma Animesh²³⁴,Slupphaug Geir²³⁴,Rolfsson Ottar¹^ORCID

Affiliation:

1. Center for Systems Biology Biomedical Center Faculty of Medicine School of Health Sciences University of Iceland Reykjavik Iceland

2. Department of Clinical and Molecular Medicine Norwegian University of Science and Technology NTNU Trondheim Norway

3. Clinic of Laboratory Medicine St. Olavs Hospital Trondheim Norway

4. PROMEC Core Facility for Proteomics and Modomics Norwegian University of Science and Technology, NTNU, and the Central Norway Regional Health Authority Norway Trondheim Norway

Funder

Icelandic Centre for Research

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.13172

Reference105 articles.

1. New insights into the mechanisms of epithelial–mesenchymal transition and implications for cancer

2. EMT in cancer

3. The EMT-activator Zeb1 is a key factor for cell plasticity and promotes metastasis in pancreatic cancer

4. Guidelines and definitions for research on epithelial–mesenchymal transition

5. ERBB2 drives YAP activation and EMT-like processes during cardiac regeneration

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