The endochondral bone protein CHM1 sustains an undifferentiated, invasive phenotype, promoting lung metastasis in Ewing sarcoma-Reference-Cited by-同舟云学术

The endochondral bone protein CHM1 sustains an undifferentiated, invasive phenotype, promoting lung metastasis in Ewing sarcoma

Published:2017-08-21 Issue:9 Volume:11 Page:1288-1301
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

von Heyking Kristina¹,Calzada-Wack Julia²,Göllner Stefanie³,Neff Frauke²,Schmidt Oxana¹,Hensel Tim¹,Schirmer David¹,Fasan Annette¹,Esposito Irene⁴,Müller-Tidow Carsten³⁵,Sorensen Poul H.⁶,Burdach Stefan¹,Richter Günther H. S.¹

Affiliation:

1. Laboratory for Functional Genomics and Transplantation Biology; Children's Cancer Research Center and Department of Pediatrics; Klinikum rechts der Isar; Technische Universität München; Comprehensive Cancer Center Munich (CCCM); German Translational Cancer Research Consortium (DKTK); Munich Germany

2. Institute of Pathology; Helmholtz Zentrum München - German Research Centre for Environmental Health (GmbH); Neuherberg Germany

3. Department of Medicine IV, Hematology and Oncology; University Hospital Halle; Germany

4. Institute of Pathology; University Düsseldorf; Germany

5. Department of Medicine V, Hematology, Oncology and Rheumatology; University of Heidelberg; Germany

6. Department of Molecular Oncology; British Columbia Cancer Research Centre; Vancouver BC Canada

Funder

Else Kröner-Fresenius-Stiftung

Wilhelm Sander-Stiftung

Bundesministerium für Bildung und Forschung

National Genome Research Network (NGFNplus)

Infrafrontier

California Department of Fish and Game