SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA ‐deficient cells-Reference-Cited by-同舟云学术

SIK2 kinase synthetic lethality is driven by spindle assembly defects in FANCA ‐deficient cells

Published:2021-06-28 Issue: Volume: Page:
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Molecular Oncology

Author:

Chan Ka‐Kui¹,Abdul‐Sater Zahi¹^ORCID,Sheth Aditya²,Mitchell Dana K.¹,Sharma Richa¹,Edwards Donna M.³,He Ying¹,Nalepa Grzegorz¹²³,Rhodes Steven D.⁴,Clapp D. Wade¹²³,Sierra Potchanant Elizabeth A.¹^ORCID

Affiliation:

1. Department of Pediatrics Riley Hospital for ChildrenIndiana University School of Medicine Indianapolis IN USA

2. Department of Medical and Molecular Genetics Indiana University School of Medicine Indianapolis IN USA

3. Department of Biochemistry and Molecular Biology Indiana University School of Medicine Indianapolis IN USA

4. Division of Pediatric Hematology‐Oncology Indiana University School of Medicine Indianapolis IN USA

Funder

National Cancer Institute

Eunice Kennedy Shriver National Institute of Child Health and Human Development

National Institutes of Health

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.13027

Reference76 articles.

1. The Fanconi Anemia/BRCA pathway: new faces in the crowd

2. Regulation of DNA cross-link repair by the Fanconi anemia/BRCA pathway

3. Fanconi anemia complementation group A (FANCA) localizes to centrosomes and functions in the maintenance of centrosome integrity

4. Fanconi anemia signaling network regulates the spindle assembly checkpoint

5. FANCA safeguards interphase and mitosis during hematopoiesis in vivo

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