Identification of BLNK and BTK as mediators of rituximab‐induced programmed cell death by CRISPR screens in GCB‐subtype diffuse large B‐cell lymphoma-Reference-Cited by-同舟云学术

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Identification of BLNK and BTK as mediators of rituximab‐induced programmed cell death by CRISPR screens in GCB‐subtype diffuse large B‐cell lymphoma

Published:2020-07-16 Issue:9 Volume:14 Page:1978-1997
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Thomsen Emil Aagaard¹,Rovsing Anne Bruun¹,Anderson Mads Valdemar¹,Due Hanne²,Huang Jinrong¹³⁴,Luo Yonglun¹³,Dybkær Karen²,Mikkelsen Jacob Giehm¹

Affiliation:

1. Department of Biomedicine Aarhus University Denmark

2. Department of Hematology Aalborg University Hospital Denmark

3. Lars Bolund Institute of Regenerative Medicine BGI‐QingdaoBGI‐Shenzhen China

4. Department of Biology University of Copenhagen Denmark

Funder

Arkitekt Holger Hjortenberg og Hustru Dagmar Hjortenbergs Fond

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.12753

Reference69 articles.

1. Lymphoma incidence patterns by WHO subtype in the United States, 1992-2001

2. Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes

3. Genetic and Functional Drivers of Diffuse Large B Cell Lymphoma

4. Genetics and Pathogenesis of Diffuse Large B-Cell Lymphoma

5. Comprehensive gene expression profiling and immunohistochemical studies support application of immunophenotypic algorithm for molecular subtype classification in diffuse large B-cell lymphoma: a report from the International DLBCL Rituximab-CHOP Consortium Program Study

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