AMPK activation induced in pemetrexed‐treated cells is associated with development of drug resistance independently of target enzyme expression-Reference-Cited by-同舟云学术

AMPK activation induced in pemetrexed‐treated cells is associated with development of drug resistance independently of target enzyme expression

Published:2019-05-15 Issue:6 Volume:13 Page:1419-1432
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Qin Yiyang¹²,Sekine Ikuo³,Hanazono Michiko¹⁴,Morinaga Takao¹,Fan Mengmeng⁵,Takiguchi Yuichi⁵,Tada Yuji⁴,Shingyoji Masato⁶,Yamaguchi Naoto²,Tagawa Masatoshi¹⁷

Affiliation:

1. Division of Pathology and Cell Therapy Chiba Cancer Center Research Institute Japan

2. Laboratory of Molecular Cell Biology Graduate School of Pharmaceutical Sciences Chiba University Japan

3. Department of Medical Oncology Faculty of Medicine University of Tsukuba Japan

4. Department of Respirology Graduate School of Medicine Chiba University Japan

5. Department of Medical Oncology Graduate School of Medicine Chiba University Japan

6. Division of Respirology Chiba Cancer Center Japan

7. Department of Molecular Biology and Oncology Graduate School of Medicine Chiba University Japan

Funder

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.12496

Reference32 articles.

1. A chemical biology approach identifies AMPK as a modulator of melanoma oncogene MITF

2. The resistance related to targeted therapy in malignant pleural mesothelioma: Why has not the target been hit yet?

3. p53 Target Genes Sestrin1 and Sestrin2 Connect Genotoxic Stress and mTOR Signaling

4. Molecular mechanism implicated in Pemetrexed-induced apoptosis in human melanoma cells

5. Pharmacological targeting of p53 through RITA is an effective antitumoral strategy for malignant pleural mesothelioma

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