High CD34 surface expression in BCP‐ALL predicts poor induction therapy response and is associated with altered expression of genes related to cell migration and adhesion-Reference-Cited by-同舟云学术

High CD34 surface expression in BCP‐ALL predicts poor induction therapy response and is associated with altered expression of genes related to cell migration and adhesion

Published:2022-04-07 Issue:10 Volume:16 Page:2015-2030
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Molecular Oncology

Author:

Modvig Signe¹²^ORCID,Wernersson Rasmus³⁴^ORCID,Øbro Nina Friesgaard¹,Olsen Lars Rønn³,Christensen Claus¹,Rosthøj Susanne⁵,Degn Matilda⁶,Jürgensen Gitte Wullf¹,Madsen Hans O.¹,Albertsen Birgitte Klug⁷⁸,Wehner Peder Skov⁹,Rosthøj Steen¹⁰,Lilljebjörn Henrik¹¹,Fioretos Thoas¹¹,Schmiegelow Kjeld²⁶,Marquart Hanne Vibeke¹

Affiliation:

1. Department of Clinical Immunology Copenhagen University Hospital Rigshospitalet Denmark

2. Faculty of Medicine Institute of Clinical Medicine University of Copenhagen Denmark

3. Section for Bioinformatics Department of Health Technology Technical University of Denmark Lyngby Denmark

4. Intomics A/S Lyngby Denmark

5. Section of Biostatistics University of Copenhagen Denmark

6. Department of Pediatric and Adolescent Medicine Copenhagen University Hospital Rigshospitalet Denmark

7. Department of Pediatrics and Adolescent Medicine Aarhus University Hospital Denmark

8. Department of Clinical Medicine Aarhus University Denmark

9. H.C. Andersen Children's Hospital Odense University Hospital Denmark

10. Department of Pediatrics and Adolescent Medicine Aalborg University Hospital Denmark

11. Division of Clinical Genetics Department of Laboratory Medicine Lund University Sweden

Funder

European Regional Development Fund

Børnecancerfonden

Kræftens Bekæmpelse

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.13207

Reference73 articles.

1. Association of Minimal Residual Disease With Clinical Outcome in Pediatric and Adult Acute Lymphoblastic Leukemia

2. Quantitative analysis of minimal residual disease predicts relapse in children with B-lineage acute lymphoblastic leukemia in DFCI ALL Consortium Protocol 95-01

3. Value of flow cytometry for MRD-based relapse prediction in B-cell precursor ALL in a multicenter setting

4. Overexpression of CD123 correlates with the hyperdiploid genotype in acute lymphoblastic leukemia

5. Human homologue of the rat chondroitin sulfate proteoglycan, NG2, detected by monoclonal antibody 7.1, identifies childhood acute lymphoblastic leukemias with t(4;11)(q21;q23) or t(11;19)(q23;p13) and MLL gene rearrangements

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