Interleukin‐22 promotes development of malignant lesions in a mouse model of spontaneous breast cancer-Reference-Cited by-同舟云学术

Interleukin‐22 promotes development of malignant lesions in a mouse model of spontaneous breast cancer

Published:2019-12-04 Issue:1 Volume:14 Page:211-224
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Katara Gajendra K.¹^ORCID,Kulshrestha Arpita¹,Schneiderman Sylvia¹,Riehl Valerie¹,Ibrahim Safaa¹²,Beaman Kenneth D.¹^ORCID

Affiliation:

1. Center for Cancer Cell Biology, Immunology and Infection Chicago Medical School Rosalind Franklin University of Medicine and Science North Chicago IL USA

2. Department of Microbiology and Immunology Faculty of Pharmacy Cairo University Egypt

Funder

National Cancer Institute

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.12598

Reference27 articles.

1. Modulation of mammary gland development in pre-pubertal mice as affected by soya and milk protein supplements

2. IL-22 is increased in active Crohn’s disease and promotes proinflammatory gene expression and intestinal epithelial cell migration

3. Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries

4. Interleukin 22 prevents lipopolysaccharide- induced preterm labor in mice†

5. Abstract 3293: Solid tumors have frequent mutation, copy number variation and differential mRNA expression of GPCRs: Are such GPCRs functional oncogenes

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