UBQLN4 is an ATM substrate that stabilizes the anti‐apoptotic proteins BCL2A1 and BCL2L10 in mesothelioma-Reference-Cited by-同舟云学术

UBQLN4 is an ATM substrate that stabilizes the anti‐apoptotic proteins BCL2A1 and BCL2L10 in mesothelioma

Published:2021-08-30 Issue:12 Volume:15 Page:3738-3752
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Liu Fang¹^ORCID,Pan RunSang²,Ding HongYu³^ORCID,Gu LiLing¹⁴⁵,Yang Yun¹,Li ChuanYin³,Xu YongJie⁵,Hu Ronggui³,Chen Hui⁵,Zhang XiangYan⁵,Nie YingJie⁵

Affiliation:

1. Medical College Guizhou University Guiyang China

2. GuiYang Maternal and Child Hospital Guiyang China

3. State Key Laboratory of Systems Biology CAS Center for Excellence in Molecular Cell Science Innovation Center for Cell Signaling Network Shanghai Institute of Biochemistry and Cell Biology Chinese Academy of Sciences Shanghai China

4. Department of Rehabilitation Guizhou Provincial People’s Hospital Guiyang China

5. NHC Key Laboratory of Pulmonary Immune‐related Diseases Guizhou Provincial People’s Hospital Guiyang China

Funder

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/1878-0261.13058

Reference41 articles.

1. The DNA Damage Response: Making It Safe to Play with Knives

2. ATM ‐ CHK 2‐Beclin 1 axis promotes autophagy to maintain ROS homeostasis under oxidative stress

3. ATM and the Molecular Pathogenesis of Ataxia Telangiectasia

4. SIRT1 modulates cell cycle progression by regulating CHK2 acetylation−phosphorylation

5. SMC1 is a downstream effector in the ATM/NBS1 branch of the human S-phase checkpoint

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