The impact of postsynaptic density 95 blocking peptide (Tat‐NR2B9c) and an iNOS inhibitor (1400W) on proteomic profile of the hippocampus in C57BL/6J mouse model of kainate‐induced epileptogenesis-Reference-Cited by-同舟云学术

The impact of postsynaptic density 95 blocking peptide (Tat‐NR2B9c) and an iNOS inhibitor (1400W) on proteomic profile of the hippocampus in C57BL/6J mouse model of kainate‐induced epileptogenesis

Published:2019-05-15 Issue:11 Volume:97 Page:1378-1392
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J Neurosci Res

Author:

Tse Karen¹²,Hammond Dean³,Simpson Deborah⁴,Beynon Robert J.⁴,Beamer Edward¹,Tymianski Michael⁵,Salter Michael W.⁵,Sills Graeme J.²,Thippeswamy Thimmasettappa¹^ORCID

Affiliation:

1. Department of Musculoskeletal Biology, Institute of Ageing and Chronic Disease University of Liverpool Liverpool UK

2. Department of Molecular and Clinical Pharmacology, Institute of Translational Medicine University of Liverpool Liverpool UK

3. Department of Molecular and Cellular Physiology, Institute of Translational Medicine University of Liverpool Liverpool UK

4. Centre for Proteome Research, Institute of Integrative Biology University of Liverpool Liverpool UK

5. Department of Physiology and Institute of Medical Sciences University of Toronto Toronto Ontario Canada

Funder

Foundation for the National Institutes of Health

University of Liverpool

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.24441

Reference74 articles.

1. Treatment of Ischemic Brain Damage by Perturbing NMDA Receptor- PSD-95 Protein Interactions

2. The Neuroprotective Effects of Heat Shock Protein 27 Overexpression in Transgenic Animals against Kainate-induced Seizures and Hippocampal Cell Death

3. Pathology and pathophysiology of the amygdala in epileptogenesis and epilepsy

4. Neuroinflammatory targets and treatments for epilepsy validated in experimental models

5. Reactive microglia in hippocampal sclerosis associated with human temporal lobe epilepsy

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