Inhibition of EZH2 Ameliorates Lupus‐Like Disease in MRL/ lpr Mice
Author:
Affiliation:
1. University of Michigan Ann Arbor
2. University of Michigan, Ann Arbor, and the Second Xiangya Hospital and Central South University Changsha China
3. University of Michigan, Ann Arbor, and University of Pittsburgh Pittsburgh Pennsylvania
Funder
National Institute of Allergy and Infectious Diseases
Lupus Research Alliance
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.40931
Reference34 articles.
1. Deoxyribonucleic Acid Methylation in Systemic Lupus Erythematosus: Implications for Future Clinical Practice
2. Updates in Lupus Genetics
3. Mechanistic aspects of epigenetic dysregulation in SLE
4. Epigenetic Variability in Systemic Lupus Erythematosus: What We Learned from Genome-Wide DNA Methylation Studies
5. Epigenetic Reprogramming in Naive CD4+ T Cells Favoring T Cell Activation and Non-Th1 Effector T Cell Immune Response as an Early Event in Lupus Flares
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1. Inducible deletion ofEzh2in CD4+ T cells inhibits kidney T cell infiltration and prevents interstitial nephritis in MRL/lprlupus-prone mice;2024-03-07
2. Prospects for the Application of Transplantation With Human Amniotic Membrane Epithelial Stem Cells in Systemic Lupus Erythematosus;Cell Transplantation;2024-01
3. Epigenetic mechanisms driving the pathogenesis of systemic lupus erythematosus, systemic sclerosis and dermatomyositis;Experimental Dermatology;2023-12-07
4. EZH2 inhibition stimulates repetitive element expression and viral mimicry in resting splenic B cells;The EMBO Journal;2023-11-07
5. Ezh2 knockout in B cells impairs plasmablast differentiation and ameliorates lupus‐like disease in MRL/lpr mice: comment on the article by Zheng et al;Arthritis & Rheumatology;2023-10-09
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