Excitatory and inhibitory neuron defects in a mouse model of Scn1b ‐linked EIEE52-Reference-Cited by-同舟云学术

Excitatory and inhibitory neuron defects in a mouse model of Scn1b ‐linked EIEE52

Published:2020-09-26 Issue:11 Volume:7 Page:2137-2149
ISSN:2328-9503
Container-title:Annals of Clinical and Translational Neurology
language:en
Short-container-title:Ann Clin Transl Neurol

Author:

Hull Jacob M.¹,O’Malley Heather A.,Chen Chunling²,Yuan Yukun²,Denomme Nicholas²,Bouza Alexandra A.²,Anumonwo Charles²,Lopez‐Santiago Luis F.²,Isom Lori L.¹²^ORCID

Affiliation:

1. Neuroscience Graduate Program University of Michigan Medical School Ann Arbor MI48109USA

2. Department of Pharmacology University of Michigan Medical School Ann Arbor MI48109USA

Funder

National Institute of Neurological Disorders and Stroke

National Institute of General Medical Sciences

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Clinical Neurology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/acn3.51205

Reference35 articles.

1. Voltage-gated sodium channels at 60: structure, function and pathophysiology

2. Voltage‐gated sodium channel beta subunits: The power outside the pore in brain development and disease;Hull JM;Neuropharmacology,2017

3. Sodium Channel β Subunits: Emerging Targets in Channelopathies

4. Not all SCN1A epileptic encephalopathies are Dravet syndrome

5. Genetic and phenotypic heterogeneity suggest therapeutic implications in SCN2A-related disorders

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