Mitochondrial dysfunction in human immunodeficiency virus‐1 transgenic mouse cardiac myocytes-Reference-Cited by-同舟云学术

Mitochondrial dysfunction in human immunodeficiency virus‐1 transgenic mouse cardiac myocytes

Published:2018-09-07 Issue:4 Volume:234 Page:4432-4444
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Cheung Joseph Y.¹²^ORCID,Gordon Jennifer³⁴,Wang JuFang¹,Song Jianliang¹,Zhang Xue‐Qian¹,Prado Fabian Jana¹,Shanmughapriya Santhanam¹⁵,Rajan Sudarsan¹⁵,Tomar Dhanendra¹⁵,Tahrir Farzaneh G.³⁴,Gupta Manish K.³⁴,Knezevic Tijana³⁴,Merabova Nana³⁴,Kontos Christopher D.⁶,McClung Joseph M.⁷,Klotman Paul E.⁸,Madesh Muniswamy¹⁵,Khalili Kamel³⁴,Feldman Arthur M.²

Affiliation:

1. Center of Translational Medicine Lewis Katz School of Medicine at Temple University Philadelphia Pennsylvania

2. Department of Medicine Lewis Katz School of Medicine at Temple University Philadelphia Pennsylvania

3. Department of Neuroscience Lewis Katz School of Medicine at Temple University Philadelphia Pennsylvania

4. Center for Neurovirology Lewis Katz School of Medicine at Temple University Philadelphia Pennsylvania

5. Department of Medical Genetics and Molecular Biochemistry Lewis Katz School of Medicine at Temple University Philadelphia Pennsylvania

6. Department of Medicine Duke University School of Medicine Durham North Carolina

7. Department of Physiology Brody School of Medicine of East Carolina University Greenville North Carolina

8. Department of Medicine Baylor College of Medicine Houston Texas

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27232

Reference45 articles.

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4. Adverse effects of reverse transcriptase inhibitors: Mitochondrial toxicity as common pathway;Brinkman K.;Acquired Immunodeficiency Syndrome,1998

5. Essential Regulation of Cell Bioenergetics by Constitutive InsP3 Receptor Ca2+ Transfer to Mitochondria

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