GPX2 silencing relieves epithelial–mesenchymal transition, invasion, and metastasis in pancreatic cancer by downregulating Wnt pathway
Author:
Affiliation:
1. Department of Imaging Linyi People's Hospital Linyi China
2. Department of Gynaecology and Obstetrics Chinese Medicine Hospital of Linyi City Linyi China
3. Department of General Surgery Linyi People's Hospital Linyi China
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.29391
Reference46 articles.
1. Inactivation of Ink4a/Arf leads to deregulated expression of miRNAs in K-Ras transgenic mouse model of pancreatic cancer
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3. β-catenin is a central mediator of pro-fibrotic Wnt signaling in systemic sclerosis
4. Pancreatic cancer genomes reveal aberrations in axon guidance pathway genes
5. Physiological functions of GPx2 and its role in inflammation-triggered carcinogenesis
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