Effects of bisphenol A on gap junctions in HaCaT cells as mediated by the estrogen receptor pathway-Reference-Cited by-同舟云学术

Effects of bisphenol A on gap junctions in HaCaT cells as mediated by the estrogen receptor pathway

Published:2018-09-06 Issue:2 Volume:39 Page:271-281
ISSN:0260-437X
Container-title:Journal of Applied Toxicology
language:en
Short-container-title:J Appl Toxicol

Author:

Zhang Qi¹^ORCID,Wu Shuang¹^ORCID,Liu Lu²,Hou Xiaohong¹,Jiang Jianjun¹,Wei Xuetao¹,Hao Weidong¹

Affiliation:

1. Department of Toxicology, School of Public Health; Peking University, Beijing Key Laboratory of Toxicological Research and Risk Assessment for Food Safety; Beijing 100191 China

2. Department of Genetics, School of Basic Medical Science; Peking University; Beijing 100191 China

Funder

Open foundation of State Key Laboratory of Environmental Chemistry and Ecotoxicology, Research Center for Eco-Environmental Sciences, Chinese Academy of Sciences

Ministry of Education of the People's Republic of China

Publisher

Wiley

Subject

Toxicology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jat.3717/fullpdf

Reference65 articles.

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4. Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line;Boukamp;The Journal of Cell Biology,1988

5. Activation of the unliganded estrogen receptor by EGF involves the MAP kinase pathway and direct phosphorylation;Bunone;The EMBO Journal,1996

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