α2AP is associated with the development of lupus nephritis through the regulation of plasmin inhibition and inflammatory responses-Reference-Cited by-同舟云学术

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α2AP is associated with the development of lupus nephritis through the regulation of plasmin inhibition and inflammatory responses

Published:2020-03-31 Issue:3 Volume:8 Page:267-278
ISSN:2050-4527
Container-title:Immunity, Inflammation and Disease
language:en
Short-container-title:Immun Inflamm Dis

Author:

Kanno Yosuke¹²^ORCID,Miyashita Mei¹,Seishima Mariko²,Matsuo Osamu³

Affiliation:

1. Department of Clinical Pathological Biochemistry, Faculty of Pharmaceutical Science Doshisha Women's College of Liberal Arts Kyoto Japan

2. Department of Dermatology Gifu University Graduate School of Medicine Gifu Japan

3. Kindai University Faculty of Medicine Osakasayama Japan

Funder

Takeda Science Foundation

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/iid3.302

Reference51 articles.

1. Pathogenesis of kidney disease in systemic lupus erythematosus

2. Mechanisms of Kidney Injury in Lupus Nephritis – the Role of Anti-dsDNA Antibodies

3. What is damaging the kidney in lupus nephritis?

4. Mechanistic connection between inflammation and fibrosis

5. The Role of Cytokine in the Lupus Nephritis

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1. The Roles of Fibrinolytic Factors in Bone Destruction Caused by Inflammation;Cells;2024-03-15

2. α2-Antiplasmin is associated with macrophage activation and fibrin deposition in a macrophage activation syndrome mouse model;Clinical and Experimental Immunology;2024-03-08

3. Exploring Systemic Lupus Erythematosus Pathogenesis through Animal Models: A Systematic Review of Humanized and Pristane-Induced Lupus Mice;Journal of Biomedicine and Translational Research;2023-12-07

4. Crosstalk between the plasminogen/plasmin system and inflammation resolution;Journal of Thrombosis and Haemostasis;2023-10

5. α2-antiplasmin is a key regulator of macrophage activation syndrome progression through modulation of IFN-γ-induced responses and fibrin deposition;2023-09-06

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