Tracer‐specific reference tissues selection improves detection of <scp> 18 F‐FDG </scp> , <scp> 18 F </scp> ‐florbetapir, and <scp> 18 F </scp> ‐flortaucipir <scp>PET SUVR</scp> changes in Alzheimer's disease-Reference-Cited by-同舟云学术

Tracer‐specific reference tissues selection improves detection of ¹⁸ F‐FDG , ¹⁸ F ‐florbetapir, and ¹⁸ F ‐flortaucipir PET SUVR changes in Alzheimer's disease

Published:2022-02-15 Issue:7 Volume:43 Page:2121-2133
ISSN:1065-9471
Container-title:Human Brain Mapping
language:en
Short-container-title:Human Brain Mapping

Author:

Li Yanxiao¹²,Ng Yee Ling¹,Paranjpe Manish D.³,Ge Qi¹,Gu Fengyun¹⁴,Li Panlong⁵,Yan Shaozhen⁶,Lu Jie⁶^ORCID,Wang Xiuying²^ORCID,Zhou Yun¹^ORCID,

Affiliation:

1. Central Research Institute United Imaging Healthcare Group Co., Ltd Shanghai China

2. School of Computer Science The University of Sydney Sydney New South Wales Australia

3. Harvard‐MIT Health Sciences and Technology Program Harvard Medical School Boston Massachusetts USA

4. Department of Statistics University College Cork Cork Ireland

5. School of Electrical and Information Engineering Zhengzhou University of Light Industry Zhengzhou Henan China

6. Department of Radiology and Nuclear Medicine, Xuanwu Hospital Capital Medical University Beijing China

Publisher

Wiley

Subject

Neurology (clinical),Neurology,Radiology, Nuclear Medicine and imaging,Radiological and Ultrasound Technology,Anatomy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hbm.25774

Reference62 articles.

1. Longitudinal PET Evaluation of Cerebral Metabolic Decline in Dementia: A Potential Outcome Measure in Alzheimer’s Disease Treatment Studies

2. Reference Tissue–Based Kinetic Evaluation of 18F-AV-1451 for Tau Imaging

3. Kinetic Modeling of the Tau PET Tracer 18F-AV-1451 in Human Healthy Volunteers and Alzheimer Disease Subjects

4. Longitudinal evaluation of surrogates of regional cerebral blood flow computed from dynamic amyloid PET imaging

5. Reference region selection and the association between the rate of amyloid accumulation over time and the baseline amyloid burden

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