Paraspeckle Protein NONO Promotes TAZ Phase Separation in the Nucleus to Drive the Oncogenic Transcriptional Program-Reference-Cited by-同舟云学术

Paraspeckle Protein NONO Promotes TAZ Phase Separation in the Nucleus to Drive the Oncogenic Transcriptional Program

Published:2021-10-29 Issue:24 Volume:8 Page:2102653
ISSN:2198-3844
Container-title:Advanced Science
language:en
Short-container-title:Advanced Science

Author:

Wei Yiju¹,Luo Huacheng¹²,Yee Patricia P.¹,Zhang Lijun³⁴,Liu Zhijun¹,Zheng Haiyan⁵,Zhang Lei¹⁶,Anderson Benjamin¹,Tang Miaolu¹,Huang Suming¹²,Li Wei¹⁴⁷^ORCID

Affiliation:

1. Division of Pediatric Hematology and Oncology Department of Pediatrics Penn State Health Hershey Medical Center Penn State College of Medicine Hershey PA 17033 USA

2. Department of Pharmacology Penn State Health Hershey Medical Center Penn State College of Medicine Hershey PA 17033 USA

3. Institute for Personalized Medicine Penn State Health Hershey Medical Center Penn State College of Medicine Hershey PA 17033 USA

4. Department of Biochemistry & Molecular Biology Penn State Health Hershey Medical Center Penn State College of Medicine Hershey PA 17033 USA

5. Biological Mass Spectrometry Facility Robert Wood Johnson Medical School, Rutgers The State University of New Jersey Piscataway NJ 08854 USA

6. Hepatic Surgery Center Tongji Hospital Tongji Medical College Huazhong University of Science and Technology Wuhan Hubei Province 430030 China

7. Penn State Cancer Institute Penn State Health Hershey Medical Center Penn State College of Medicine Hershey PA 17033 USA

Funder

National Cancer Institute

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

General Physics and Astronomy,General Engineering,Biochemistry, Genetics and Molecular Biology (miscellaneous),General Materials Science,General Chemical Engineering,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/advs.202102653

Reference45 articles.

1. Hippo Pathway in Organ Size Control, Tissue Homeostasis, and Cancer

2. Transcriptional addiction in cancer cells is mediated by YAP/TAZ through BRD4

3. Genome-wide association between YAP/TAZ/TEAD and AP-1 at enhancers drives oncogenic growth

4. YAP Drives Growth by Controlling Transcriptional Pause Release from Dynamic Enhancers

5. YAP1 Exerts Its Transcriptional Control via TEAD-Mediated Activation of Enhancers

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