Remediating Desmoplasia with EGFR‐Targeted Photoactivable Multi‐Inhibitor Liposomes Doubles Overall Survival in Pancreatic Cancer-Reference-Cited by-同舟云学术

Remediating Desmoplasia with EGFR‐Targeted Photoactivable Multi‐Inhibitor Liposomes Doubles Overall Survival in Pancreatic Cancer

Published:2022-06-24 Issue:24 Volume:9 Page:2104594
ISSN:2198-3844
Container-title:Advanced Science
language:en
Short-container-title:Advanced Science

Author:

Obaid Girgis¹^ORCID,Bano Shazia¹^ORCID,Thomsen Hanna¹,Callaghan Susan¹,Shah Nimit²,Swain Joseph W. R.¹^ORCID,Jin Wendong¹^ORCID,Ding Xiadong¹,Cameron Colin G.³,McFarland Sherri A.³,Wu Juwell¹,Vangel Mark¹^ORCID,Stoilova‐McPhie Svetla⁴^ORCID,Zhao Jie¹,Mino‐Kenudson Mari¹^ORCID,Lin Charles¹^ORCID,Hasan Tayyaba¹⁵^ORCID

Affiliation:

1. Department of Dermatology Massachusetts General Hospital and Harvard Medical School Boston MA 02114 USA

2. Department of Bioengineering University of Texas at Dallas Richardson TX 75080 USA

3. University of Texas at Arlington Arlington TX 76019 USA

4. Center for Nanoscale Systems Harvard University Cambridge MA 02138 USA

5. Division of Health Sciences and Technology Harvard University and Massachusetts Institute of Technology Cambridge MA 02139 USA

Funder

National Institutes of Health

Science Foundation Ireland

Irish Research Council

Publisher

Wiley

Subject

General Physics and Astronomy,General Engineering,Biochemistry, Genetics and Molecular Biology (miscellaneous),General Materials Science,General Chemical Engineering,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/advs.202104594

Reference68 articles.

1. Epidemiology of Pancreatic Cancer: Global Trends, Etiology and Risk Factors

2. Prevailing Role of Contact Guidance in Intrastromal T-cell Trapping in Human Pancreatic Cancer

3. Inhibition of Hedgehog Signaling Enhances Delivery of Chemotherapy in a Mouse Model of Pancreatic Cancer

4. Targeting Tumor Architecture to Favor Drug Penetration: A New Weapon to Combat Chemoresistance in Pancreatic Cancer?

5. Proteomic analyses of ECM during pancreatic ductal adenocarcinoma progression reveal different contributions by tumor and stromal cells

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