Panax notoginseng saponins suppress lipopolysaccharide‐induced barrier disruption and monocyte adhesion on bEnd.3 cells via the opposite modulation of Nrf2 antioxidant and NF‐κB inflammatory pathways-Reference-Cited by-同舟云学术

Panax notoginseng saponins suppress lipopolysaccharide‐induced barrier disruption and monocyte adhesion on bEnd.3 cells via the opposite modulation of Nrf2 antioxidant and NF‐κB inflammatory pathways

Published:2019-08-30 Issue:12 Volume:33 Page:3163-3176
ISSN:0951-418X
Container-title:Phytotherapy Research
language:en
Short-container-title:Phytotherapy Research

Author:

Hu Shaonan¹^ORCID,Liu Tingting²,Wu Yali¹,Yang Wanqing¹,Hu Shaobo³,Sun Zongxi¹,Li Pengyue¹,Du Shouying¹

Affiliation:

1. School of Chinese Materia MedicaBeijing University of Chinese Medicine Beijing China

2. State Key Laboratory of Natural MedicinesChina Pharmaceutical University Nanjing China

3. School of Clinical Chinese MedicineBeijing University of Chinese Medicine Beijing China

Funder

Fundamental Research Funds for the Central Universities

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ptr.6488

Reference44 articles.

1. Neuroprotective effects of vildagliptin in rat rotenone Parkinson's disease model: role of RAGE-NFκB and Nrf2-antioxidant signaling pathways

2. Interactions of Oxidative Stress and Neurovascular Inflammation in the Pathogenesis of Traumatic Brain Injury

3. Limiting inflammation—the negative regulation of NF-κB and the NLRP3 inflammasome

4. Nrf2 signaling pathway: Pivotal roles in inflammation

5. Blockade of Apoptosis Signal-Regulating Kinase 1 Attenuates Matrix Metalloproteinase 9 Activity in Brain Endothelial Cells and the Subsequent Apoptosis in Neurons after Ischemic Injury

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