Variants on the UBE2L3 / YDJC Autoimmune Disease Risk Haplotype Increase UBE2L3 Expression by Modulating CCCTC‐Binding Factor and YY1 Binding-Reference-Cited by-同舟云学术

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Variants on the UBE2L3 / YDJC Autoimmune Disease Risk Haplotype Increase UBE2L3 Expression by Modulating CCCTC‐Binding Factor and YY1 Binding

Published:2021-12-13 Issue:1 Volume:74 Page:163-173
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Gopalakrishnan Jaanam¹,Tessneer Kandice L.²,Fu Yao²,Pasula Satish²,Pelikan Richard C.²,Kelly Jennifer A.²,Wiley Graham B.²,Gaffney Patrick M.¹^ORCID

Affiliation:

1. Oklahoma Medical Research Foundation and University of Oklahoma Health Sciences Center Oklahoma City

2. Oklahoma Medical Research Foundation Oklahoma City

Funder

National Institute of General Medical Sciences

National Institute of Arthritis and Musculoskeletal and Skin Diseases

National Institute of Allergy and Infectious Diseases

Presbyterian Health Foundation

NIH

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.41925

Reference41 articles.

1. Linear ubiquitin chains: enzymes, mechanisms and biology

2. Linear polyubiquitin chains

3. Involvement of linear polyubiquitylation of NEMO in NF-κB activation

4. Mechanism Underlying IκB Kinase Activation Mediated by the Linear Ubiquitin Chain Assembly Complex

5. UBE2L3 Polymorphism Amplifies NF-κB Activation and Promotes Plasma Cell Development, Linking Linear Ubiquitination to Multiple Autoimmune Diseases

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