Dichlorodiphenyldichloroethylene exposure reduces r-GCS via suppressed Nrf2 in HepG2 cells

Author:

Jin Xiaoting1,Song Li12,Li Zhuoyu13,Newton Ian P.4,Zhao Meirong5,Liu Weiping2

Affiliation:

1. Institute of Biotechnology, Key Laboratory of Chemical Biology and Molecular Engineering of National Ministry of Education; Shanxi University; Taiyuan 030006 China

2. MOE Key Lab of Environmental Remediation and Ecosystem Health, College of Environmental and Resource Sciences; Zhejiang University; 866 Yuhangtang Road Hangzhou 310058 China

3. College of Life Science; Zhejiang Chinese Medical University; Hangzhou 310053 China

4. Division of Cell & Developmental Biology, College of Life Sciences; University of Dundee; Dundee DD15EH Scotland UK

5. Research Center of Environmental Science; Zhejiang University of Technology; Hangzhou 310032 China

Funder

National Natural Sciences Foundation of China

Natural Science Foundation of Shanxi

Research Fund for the Doctoral Program of Higher Education of China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Reference53 articles.

1. Pesticides and oxidative stress: A review;Abdollahi;Med Sci Monit,2004

2. Altered lipid profile, oxidative status and hepatitis B virus interactions in human hepatocellular carcinoma;Abel;Prostaglandins Leukot Essent Fatty Acids,2009

3. Pesticide-induced oxidative stress: Perspective and trends;Banerjee;Rev Environ Health,2001

4. Role of glutathione and other thiols in cellular response to radiation and drugs;Biaglow;Drug Metab Rev,1989

5. Protein oxidation and cellular homeostasis: Emphasis on metabolism;Cecarini;Biochim Biophys Acta,2007

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