IL-33 facilitates oncogene-induced cholangiocarcinoma in mice by an interleukin-6-sensitive mechanism

Author:

Yamada Daisaku1,Rizvi Sumera1,Razumilava Nataliya1,Bronk Steven F.1,Davila Jaime I.2,Champion Mia D.3,Borad Mitesh J.4,Bezerra Jorge A.5,Chen Xin6,Gores Gregory J.1

Affiliation:

1. Division of Gastroenterology and Hepatology; Mayo Clinic; Rochester MN

2. Department of Health Sciences Research; Mayo Clinic; Rochester MN

3. Department of Biomedical Statistics and Informatics; Mayo Clinic; Scottsdale Arizona

4. Division of Hematology/Oncology; Mayo Clinic; Scottsdale AZ

5. Division of Gastroenterology, Hepatology and Nutrition; Cincinnati Children's Hospital Medical Center; Cincinnati OH

6. Department of Bioengineering and Therapeutic Sciences; University of California; San Francisco CA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Hepatology

Reference32 articles.

1. Pathogenesis, diagnosis, and management of cholangiocarcinoma;Rizvi;Gastroenterology,2013

2. Risk factors for cholangiocarcinoma;Tyson;Hepatology,2011

3. Integrative molecular analysis of intrahepatic cholangiocarcinoma reveals 2 classes that have different outcomes;Sia;Gastroenterology,2013

4. The role of cancer-associated myofibroblasts in intrahepatic cholangiocarcinoma;Sirica;Nat Rev Gastroenterol Hepatol,2012

5. Burden of digestive diseases in the United States;Everhart;Part III: Liver, biliary tract, and pancreas. Gastroenterology,2009

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