MicroRNA ‐155 deficiency attenuates inflammation and oxidative stress in experimental autoimmune prostatitis in a TLR4 ‐dependent manner-Reference-Cited by-同舟云学术

MicroRNA ‐155 deficiency attenuates inflammation and oxidative stress in experimental autoimmune prostatitis in a TLR4 ‐dependent manner

Published:2020-05-21 Issue:9 Volume:36 Page:712-720
ISSN:1607-551X
Container-title:The Kaohsiung Journal of Medical Sciences
language:en
Short-container-title:The Kaohsiung Journal of Medical Sciences

Author:

Fu Xian¹,He Hua‐Dong¹,Li Chang‐Jiu²,Li Ning¹,Jiang Shu‐Yuan²,Ge Hong‐Wei¹,Wang Rui²,Wang Xu‐Liang¹^ORCID

Affiliation:

1. Department of Urology, Affiliated Hangzhou First People's Hospital, School of Medicine Zhejiang University Zhejiang Hangzhou China

2. Department of Urology Nanjing Medical University Nanjing Jiangsu China

Funder

Science and Technology Planning Project of Zhejiang Province

Publisher

Wiley

Subject

General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/kjm2.12229

Reference37 articles.

1. Experimental rodent models of chronic prostatitis and evaluation criteria

2. A nanoparticle‐coupled T2 peptide induces immune tolerance and ameliorates chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS) in mice model

3. Effect of Shirexiao waist hot‐compress on the expressions of Th17/Treg‐specific factors in the mouse model of experimental autoimmune prostatitis with damp heat syndrome;Zhu M;Natl J Androl,2017

4. Latency Locus Complements MicroRNA 155 Deficiency In Vivo

5. Under‐expression of microRNA‐155 may inhibit prostate growth through SMAD‐2 pathway in patients with benign prostatic hyperplasia;Antunes AA;J Urol,1742

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