Mitochondrial DNA, nuclear context, and the risk for carcinogenesis-Reference-Cited by-同舟云学术

Mitochondrial DNA, nuclear context, and the risk for carcinogenesis

Published:2018-01-14 Issue:5 Volume:60 Page:455-462
ISSN:0893-6692
Container-title:Environmental and Molecular Mutagenesis
language:en
Short-container-title:Environ Mol Mutagen

Author:

Kaufman Brett A.¹,Picard Martin²³⁴,Sondheimer Neal⁵⁶^ORCID

Affiliation:

1. Center for Metabolism and Mitochondrial Medicine, Division of CardiologyVascular Medicine Institute, Department of Medicine, University of Pittsburgh Medical SchoolPittsburgh Pennsylvania

2. Department of Psychiatry, Division of Behavioral MedicineColumbia University Medical CenterNew York New York

3. Department of Neurology, H. Houston Merritt Center, Columbia Translational Neuroscience InitiativeColumbia University Medical CenterNew York New York

4. Columbia Aging Center, Columbia University Mailman School of Public HealthNew York New York

5. Division of Clinical and Metabolic GeneticsThe Hospital for Sick ChildrenToronto OntarioM5G1X8 Canada

6. Department of PaediatricsThe University of Toronto School of MedicineToronto OntarioM5G1X8 Canada

Funder

National Institutes of Health

March of Dimes

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Genetics(clinical),Epidemiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/em.22169

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5. An essential role of the mitochondrial electron transport chain in cell proliferation is to enable aspartate synthesis;Birsoy K;Cell,2015

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