MAPK pathway‐activating mutations drive giant cell lesions of the jaws and non‐ossifying fibromas of bone
Author:
Affiliation:
1. Department of Pathology, Biological Sciences InstituteUniversidade Federal de Minas Gerais Belo Horizonte Brazil
2. Department of Oral Surgery and Pathology, Faculty of DentistryUniversidade Federal de Minas Gerais Belo Horizonte Brazil
Publisher
Wiley
Subject
Pathology and Forensic Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/path.5235
Reference8 articles.
1. Activating mutations in the MAP‐kinase pathway define non‐ossifying fibroma of bone
2. Distinct H3F3A and H3F3B driver mutations define chondroblastoma and giant cell tumor of bone
3. The highly prevalent H3F3A mutation in giant cell tumours of bone is not shared by sporadic central giant cell lesion of the jaws
4. TRPV4 and KRAS and FGFR1 gain-of-function mutations drive giant cell lesions of the jaw
5. Oculoectodermal syndrome is a mosaic RASopathy associated withKRASalterations
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1. DNA methylation profile discriminates sporadic giant cell granulomas of the jaws and cherubism from their giant cell‐rich histological mimics;The Journal of Pathology: Clinical Research;2023-08-09
2. Edema-like marrow signal intensity (ELMSI) associated with nonossifying fibroma (NOF) on MRI: an uncommon finding in a common bone lesion;Skeletal Radiology;2023-03-03
3. Oestrogen receptor expression distinguishes non-ossifying fibroma from other giant cell containing bone tumours;Virchows Archiv;2022-05-25
4. Granulome central à cellules géantes des maxillaires bilatéral dans le cadre d’un syndrome de Noonan : à propos d’un cas avec mise au point sur les lésions osseuses riches en cellules géantes des maxillaires;Annales de Pathologie;2022-04
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