Regulation of S1PR2 by the EBV oncogene LMP1 in aggressive ABC‐subtype diffuse large B‐cell lymphoma-Reference-Cited by-同舟云学术

Regulation of S1PR2 by the EBV oncogene LMP1 in aggressive ABC‐subtype diffuse large B‐cell lymphoma

Published:2019-03-22 Issue:2 Volume:248 Page:142-154
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol.

Author:

Vockerodt Martina¹²,Vrzalikova Katerina¹^ORCID,Ibrahim Maha¹³,Nagy Eszter¹,Margielewska Sandra¹,Hollows Robert¹,Lupino Lauren¹,Tooze Reuben⁴^ORCID,Care Matthew⁴,Simmons William¹,Schrader Alexandra¹²⁵,Perry Tracey¹,Abdullah Maizaton¹⁶,Foster Stephen¹,Reynolds Gary⁷,Dowell Alexander¹⁷,Rudzki Zbigniew⁸,Krappmann Daniel⁹,Kube Dieter⁵,Woodman Ciaran¹,Wei Wenbin¹¹⁰,Taylor Graham⁷,Murray Paul G¹¹¹

Affiliation:

1. Institute of Cancer and Genomic SciencesUniversity of Birmingham Birmingham UK

2. Department of Anatomy and Cell Biology, University Medical CentreGeorg‐August University of Göttingen Göttingen Germany

3. South Egypt Cancer InstituteAssiut University Assiut Egypt

4. Leeds Institute of Cancer and PathologyUniversity of Leeds Leeds UK

5. Department of Hematology & Oncology and GRK 1034 of the Deutsche ForschungsgemeinschaftGeorg‐August University of Göttingen Göttingen Germany

6. Department of PathologyUniversiti Putra Malaysia Seri Kembangan Malaysia

7. Institute of Immunology and ImmunotherapyUniversity of Birmingham Birmingham UK

8. Department of HistopathologyHeartlands Hospital Birmingham UK

9. Research Unit Cellular Signal IntegrationHelmholtz Zentrum München Neuherberg Germany

10. Sheffield Institute of Translational NeuroscienceUniversity of Sheffield Sheffield UK

11. Department of Clinical and Molecular PathologyInstitute of Molecular and Translational Medicine, Palacky University Olomouc Czech Republic

Funder

Bloodwise

Publisher

Wiley

Subject

Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/path.5237

Reference62 articles.

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4. Sensitivity to PI3K and AKT inhibitors is mediated by divergent molecular mechanisms in subtypes of DLBCL

5. Senile EBV+ B-Cell Lymphoproliferative Disorders

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