BCL2L2 loss renders ‐14q renal cancer dependent on BCL2L1 that mediates resistance to tyrosine kinase inhibitors
Author:
Affiliation:
1. Department of Urology Huashan Hospital Fudan University Shanghai P. R. China
2. Institute of Urology Fudan University Shanghai P. R. China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Molecular Medicine,Medicine (miscellaneous)
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/ctm2.348
Reference10 articles.
1. Deletions of chromosomes 3p and 14q molecularly subclassify clear cell renal cell carcinoma
2. Comprehensive analysis of copy number variance and sensitivity to common targeted therapy in clear cell renal cell carcinoma: In silico analysis with in vitro validation
3. Association between copy‐number alteration of +20q, −14q and −18p and cross‐sensitivity to tyrosine kinase inhibitors in clear‐cell renal cell carcinoma;Wang L;Cancer Cell International,2020
4. HIF1α is not a target of 14q deletion in clear cell renal cancer;Shenoy N;Scientific Reports,2020
5. Integrative Analysis of Complex Cancer Genomics and Clinical Profiles Using the cBioPortal
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1. Correction to: BCL2L2 loss renders ‐14q renal cancer dependent on BCL2L1 that mediates resistance to tyrosine kinase inhibitors;Clinical and Translational Medicine;2023-05
2. Arborinine from Glycosmis parva leaf extract inhibits clear-cell renal cell carcinoma by inhibiting KDM1A/UBE2O signaling;Food & Nutrition Research;2022-09-16
3. PFKFB4 is overexpressed in clear-cell renal cell carcinoma promoting pentose phosphate pathway that mediates Sunitinib resistance;Journal of Experimental & Clinical Cancer Research;2021-09-30
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