Functional role of the KCa3.1 potassium channel in synovial fibroblasts from rheumatoid arthritis patients
Author:
Affiliation:
1. Center for Molecular Biomedicine, Department of Biophysics; Friedrich Schiller University of Jena and Jena University Hospital; Jena Germany
2. Experimental Rheumatology Unit, Department of Orthopedics; Jena University Hospital; Jena Germany
Funder
German Federal Ministry of Education and Research
German Research Foundation
Deutsche Krebshilfe e.V. (German Cancer Aid)
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcp.24924/fullpdf
Reference39 articles.
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2. Efficacy and safety of the Gardos channel blocker, senicapoc (ICA-17043), in patients with sickle cell anemia;Ataga;Blood,2008
3. The membrane potentials of fibroblasts in different environments;Bard;J Cell Physiol,1974
4. Fibroblast-like synoviocytes: Key effector cells in rheumatoid arthritis;Bartok;Immunol Rev,2010
5. Physiological roles of the intermediate conductance, Ca2+ -activated potassium channel Kcnn4;Begenisich;J Biol Chem,2004
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1. Regulatory role of KCa3.1 in immune cell function and its emerging association with rheumatoid arthritis;Frontiers in Immunology;2022-10-05
2. Dextran sodium sulfate potentiates NLRP3 inflammasome activation by modulating the KCa3.1 potassium channel in a mouse model of colitis;Cellular & Molecular Immunology;2022-07-07
3. Pro-inflammatory Cytokines Drive Deregulation of Potassium Channel Expression in Primary Synovial Fibroblasts;Frontiers in Physiology;2020-03-24
4. Pro-inflammatory cytokines drive deregulation of potassium channel expression in primary synovial fibroblasts;2020-02-07
5. Ca2+signalling in fibroblasts and the therapeutic potential of KCa3.1 channel blockers in fibrotic diseases;British Journal of Pharmacology;2020-02-03
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