Repression of TXNIP–NLRP3 axis restores intestinal barrier function via inhibition of myeloperoxidase activity and oxidative stress in nonalcoholic steatohepatitis-Reference-Cited by-同舟云学术

Repression of TXNIP–NLRP3 axis restores intestinal barrier function via inhibition of myeloperoxidase activity and oxidative stress in nonalcoholic steatohepatitis

Published:2018-11 Issue:5 Volume:234 Page:7524-7538
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Bai Ru‐Xue¹,Xu Ying‐Ying¹,Qin Geng¹,Chen Yan‐Ming¹,Wang Hui‐Fen¹,Wang Miao¹,Du Shi‐Yu¹^ORCID

Affiliation:

1. Department of Gastroenterology China‐Japan Friendship Hospital Beijing China

Funder

China-Japan Friendship Hospital Youth Science and Technology Excellence Project

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27513

Reference43 articles.

1. Histone deacetylase 3 coordinates commensal-bacteria-dependent intestinal homeostasis

2. Melatonin alleviates cadmium-induced liver injury by inhibiting the TXNIP-NLRP3 inflammasome

3. Early Detection of Nonalcoholic Steatohepatitis in Patients with Nonalcoholic Fatty Liver Disease by Using MR Elastography

4. Activation of the TXNIP/NLRP3 inflammasome pathway contributes to inflammation in diabetic retinopathy: a novel inhibitory effect of minocycline

5. Critical role of TXNIP in oxidative stress, DNA damage and retinal pericyte apoptosis under high glucose: Implications for diabetic retinopathy

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