High glucose-mediated alterations of mechanisms important in myogenesis of mouse C2C12 myoblasts
Author:
Affiliation:
1. Department of Physiological Sciences, Faculty of Veterinary Medicine; Warsaw University of Life Sciences (SGGW); Nowoursynowska 159, 02-776 Warsaw Poland
Publisher
Wiley
Subject
Cell Biology,General Medicine
Reference28 articles.
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2. Cross-talk between GlcNAcylation and phosphorylation: roles in insulin resistance and glucose toxicity;Copeland;Am J Physiol Endocrinol Metab,2008
3. Involvement of myogenic regulator factors during fusion in the cell line C2C12;Dedieu;Int J Dev Biol,2002
4. Expression and subcellular localization of myogenic regulatory factors during the differentiation of skeletal muscle C2C12 myoblasts;Ferri;J Cell Biochem,2009
5. The impairment of IGF-I-stimulated protein synthesis and activation of protein kinase B, p70S6k, MAP kinase, and p90rsk in mouse C2C12 myogenic cells exposed to high glucose and high insulin;Grzelkowska-Kowalczyk;Pol J Vet Sci,2005
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