Chronic coexposure to arsenic and estrogen potentiates genotoxic estrogen metabolic pathway and hypermethylation of DNA glycosylase <i>MBD4</i> in human prostate epithelial cells-Reference-Cited by-同舟云学术

Chronic coexposure to arsenic and estrogen potentiates genotoxic estrogen metabolic pathway and hypermethylation of DNA glycosylase MBD4 in human prostate epithelial cells

Published:2022-06-23 Issue:13 Volume:82 Page:1273-1283
ISSN:0270-4137
Container-title:The Prostate
language:en
Short-container-title:The Prostate

Author:

Treas Justin¹,Roy Priti²,Singh Kamaleshwar P.³^ORCID

Affiliation:

1. Department of Internal Medicine University of Arkansas for Medical Sciences Little Rock Arkansas USA

2. Department of Internal Medicine University of California San Francisco San Francisco California USA

3. Department of Environmental Toxicology, The Institute of Environmental and Human Health (TIEHH) Texas Tech University Lubbock Texas USA

Publisher

Wiley

Subject

Urology,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/pros.24401

Reference58 articles.

1. Epidemiology of prostate cancer

2. Environmental causes of cancer: endocrine disruptors as carcinogens

3. Estrogenic chemicals in plastic and oral contraceptives disrupt development of the fetal mouse prostate and urethra

4. Developmental estrogen exposures predispose to prostate carcinogenesis with aging☆

5. Induction at high incidence of ductal prostate adenocarcinomas in NBL/Cr and Sprague-Dawley Hsd:SD rats treated with a combination of testosterone and estradiol-17β or diethylstilbestrol

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