Lack of association between bridging integrator 1 ( <i>BIN1</i> ) rs744373 polymorphism and tau‐PET load in cognitively intact older adults-Reference-Cited by-同舟云学术

Lack of association between bridging integrator 1 ( BIN1 ) rs744373 polymorphism and tau‐PET load in cognitively intact older adults

Published:2022-01 Issue:1 Volume:8 Page:
ISSN:2352-8737
Container-title:Alzheimer's & Dementia: Translational Research & Clinical Interventions
language:en
Short-container-title:A&D Transl Res & Clin Interv

Author:

Schaeverbeke Jolien¹²,Luckett Emma S¹,Gabel Silvy¹,Reinartz Mariska¹,De Meyer Steffi³,Cleynen Isabelle⁴,Sleegers Kristel⁵⁶,Van Broeckhoven Christine⁵⁶,Bormans Guy⁷,Serdons Kim⁸,Van Laere Koen⁸⁹,Dupont Patrick¹,Vandenberghe Rik¹¹⁰,

Affiliation:

1. Department of Neurosciences, Laboratory for Cognitive Neurology Leuven Brain Institute, KU Leuven Leuven Belgium

2. Department of Imaging and Pathology, Laboratory of Neuropathology Leuven Brain Institute, KU Leuven Leuven Belgium

3. Department of Neurosciences, Laboratory for Molecular Neurobiomarker Research Leuven Brain Institute, KU Leuven Leuven Belgium

4. Laboratory for Complex Genetics KU Leuven Leuven Belgium

5. VIB‐UAntwerp Center for Molecular Neurology Antwerp Belgium

6. Department of Biomedical Sciences University of Antwerp Antwerp Belgium

7. Laboratory for Radiopharmaceutical Research KU Leuven Leuven Belgium

8. Division of Nuclear Medicine UZ Leuven Leuven Belgium

9. Department of Imaging and Pathology, Nuclear Medicine and Molecular Imaging KU Leuven Leuven Belgium

10. Department of Neurology UZ Leuven Leuven Belgium

Publisher

Wiley

Subject

Psychiatry and Mental health,Neurology (clinical)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/trc2.12227

Reference31 articles.

1. Genome-wide Analysis of Genetic Loci Associated With Alzheimer Disease

2. Replication of BIN1 Association with Alzheimer's Disease and Evaluation of Genetic Interactions

3. Genome-Wide Association of Familial Late-Onset Alzheimer's Disease Replicates BIN1 and CLU and Nominates CUGBP2 in Interaction with APOE

4. Increased expression of BIN1 mediates Alzheimer genetic risk by modulating tau pathology

5. Subcellular Changes in Bridging Integrator 1 Protein Expression in the Cerebral Cortex During the Progression of Alzheimer Disease Pathology

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