Exposure interval to ambient fine particulate matter (PM2.5) collected in Southwest China induced pulmonary damage through the Janus tyrosine protein kinase‐2/signal transducer and activator of transcription‐3 signaling pathway both in vivo and in vitro-Reference-Cited by-同舟云学术

Exposure interval to ambient fine particulate matter (PM2.5) collected in Southwest China induced pulmonary damage through the Janus tyrosine protein kinase‐2/signal transducer and activator of transcription‐3 signaling pathway both in vivo and in vitro

Published:2021-06-03 Issue:12 Volume:41 Page:2042-2054
ISSN:0260-437X
Container-title:Journal of Applied Toxicology
language:en
Short-container-title:J Appl Toxicol

Author:

Yue Wuyang¹²^ORCID,Chen Xuxi¹,He Sifu³,Li Na¹,Zhang Lishi¹,Chen Jinyao¹^ORCID

Affiliation:

1. West China School of Public Health and West China Fourth Hospital Sichuan University Chengdu China

2. Department of Tuberculosis Institute Research Chongqing Public Health Medical Center/Public Health Hospital Affiliated to Southwest University Chongqing China

3. Administration Department Sichuan Kangchen Biotechnology Co. Chengdu China

Publisher

Wiley

Subject

Toxicology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jat.4196

Reference43 articles.

1. Chronic obstructive pulmonary disease: molecular and cellularmechanisms

2. Associations of PM 2.5 Constituents and Sources with Hospital Admissions: Analysis of Four Counties in Connecticut and Massachusetts (USA) for Persons ≥ 65 Years of Age

3. Air pollution and retained particles in the lung.

4. Urban airborne particulate: genotoxicity evaluation of different size fractions by mutagenesis tests on microorganisms and comet assay

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