The 28‐amino acid form of an APLP1‐derived Aβ‐like peptide is a surrogate marker for Aβ42 production in the central nervous system-Reference-Cited by-同舟云学术

The 28‐amino acid form of an APLP1‐derived Aβ‐like peptide is a surrogate marker for Aβ42 production in the central nervous system

Published:2009-07 Issue:4 Volume:1 Page:223-235
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Yanagida Kanta¹,Okochi Masayasu¹,Tagami Shinji¹,Nakayama Taisuke¹,Kodama Takashi S.¹,Nishitomi Kouhei¹,Jiang Jingwei¹,Mori Kohji¹,Tatsumi Shin‐ichi¹,Arai Tetsuaki²,Ikeuchi Takeshi³,Kasuga Kensaku³,Tokuda Takahiko⁴,Kondo Masaki⁴,Ikeda Masaki⁵,Deguchi Kentaro⁶,Kazui Hiroaki¹,Tanaka Toshihisa¹,Morihara Takashi¹,Hashimoto Ryota¹,Kudo Takashi¹,Steiner Harald⁷,Haass Christian⁷,Tsuchiya Kuniaki²,Akiyama Haruhiko²,Kuwano Ryozo⁸,Takeda Masatoshi¹

Affiliation:

1. Psychiatry, Department of Integrated Medicine, Division of Internal Medicine, Osaka University Graduate School of Medicine, Osaka 565‐0871, Japan

2. Department of Psychogeriatrics, Tokyo Institute of Psychiatry, Tokyo 156‐8585, Japan

3. Department of Molecular Neuroscience, Brain Research Institute, Niigata University, Niigata 951‐8585, Japan

4. Department of Neurology and Gerontology, Kyoto Prefectural University of Medicine, Kyoto 602‐8566, Japan

5. Department of Neurology, Gunma University School of Medicine, Gunma 371‐8511, Japan

6. Department of Neurology, Neuroscience, Biophysiological Science, Okayama University Graduate School of Medicine, Dentistry and Pharmacy, Okayama 700‐8558, Japan

7. Department of Biochemistry, Ludwig‐Maximilians‐University‐Munich, Munich D‐80336, Germany

8. Department of Molecular genetics, Center for Bioresources, Brain Research Institute, Niigata University, Niigata 951‐8585, Japan

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/emmm.200900026

Reference41 articles.

1. Cerebrospinal Fluid β-Amyloid(1-42) in Alzheimer Disease

2. Coordinated Metabolism of Alcadein and Amyloid β-Protein Precursor Regulates FE65-dependent Gene Transactivation

3. Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo