A population of naive‐like CD4<sup>+</sup>T cells stably polarized to the T<sub>H</sub>1 lineage-Reference-Cited by-同舟云学术

A population of naive‐like CD4⁺T cells stably polarized to the T_H1 lineage

Published:2022-02-12 Issue:4 Volume:52 Page:566-581
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur J Immunol

Author:

Lo Jonathan W.¹²^ORCID,de Mucha Maria Vila³,Henderson Stephen³,Roberts Luke B.¹,Constable Laura E.¹²,Garrido‐Mesa Natividad¹⁴,Hertweck Arnulf³,Stolarczyk Emilie⁵⁶,Houlder Emma L.⁷,Jackson Ian¹,MacDonald Andrew S.⁷,Powell Nick¹²,Neves Joana F.¹⁸,Howard Jane K.⁶,Jenner Richard G.³,Lord Graham M.¹⁷⁹

Affiliation:

1. School of Immunology and Microbial Sciences King's College London London UK

2. Division of Digestive Diseases Faculty of Medicine Imperial College London London UK

3. UCL Cancer Institute and CRUK UCL Centre University College London (UCL) London UK

4. School of Life Sciences, Pharmacy and Chemistry Kingston University London UK

5. Abcam Plc. Cambridge Biomedical Campus Cambridge UK

6. School of Cardiovascular Medicine and Sciences Guy's Campus, King's College London London UK

7. Lydia Becker Institute of Immunology and Inflammation, Faculty of Biology, Medicine and Health University of Manchester Manchester UK

8. Centre for Host‐Microbiome Interactions King's College London London UK

9. School of Biological Sciences, Faculty of Biology, Medicine and Health University of Manchester Manchester UK

Funder

Medical Research Council

Wellcome Trust

Cancer Research UK

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/eji.202149228

Reference79 articles.

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2. A Novel Transcription Factor, T-bet, Directs Th1 Lineage Commitment

3. T-bet: a bridge between innate and adaptive immunity

4. The transcription factors T-bet and GATA-3 control alternative pathways of T-cell differentiation through a shared set of target genes

5. T-bet is required for optimal proinflammatory CD4+ T-cell trafficking

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