Pim Kinases as Therapeutic Targets in Early Rheumatoid Arthritis-Reference-Cited by-同舟云学术

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Pim Kinases as Therapeutic Targets in Early Rheumatoid Arthritis

Published:2021-08-24 Issue:10 Volume:73 Page:1820-1830
ISSN:2326-5191
Container-title:Arthritis & Rheumatology
language:en
Short-container-title:Arthritis & Rheumatology

Author:

Maney Nicola J.¹^ORCID,Lemos Henrique¹,Barron‐Millar Ben¹,Carey Christopher¹,Herron Ian¹,Anderson Amy E.¹,Mellor Andrew L.¹,Isaacs John D.²,Pratt Arthur G.²^ORCID

Affiliation:

1. Newcastle University Translational and Clinical Research Institute Newcastle University Newcastle upon Tyne UK

2. Newcastle University Translational and Clinical Research Institute Newcastle University, and Newcastle upon Tyne Hospitals NHS Foundation Trust Newcastle upon Tyne UK

Funder

National Institute for Health Research

JGW Patterson Foundation

Versus Arthritis

Publisher

Wiley

Subject

Immunology,Rheumatology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.41744

Reference50 articles.

1. PIM kinases: From survival factors to regulators of cell motility

2. UniProt: the universal protein knowledgebase

3. Pim kinases phosphorylate multiple sites on Bad and promote 14-3-3 binding and dissociation from Bcl-XL

4. Pim-1 Kinase-Dependent Phosphorylation of p21Cip1/WAF1Regulates Its Stability and Cellular Localization in H1299 Cells

5. Pim Kinases Promote Cell Cycle Progression by Phosphorylating and Down-regulating p27Kip1 at the Transcriptional and Posttranscriptional Levels

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