Longitudinal evaluation of iron concentration and atrophy in the dentate nuclei in friedreich ataxia-Reference-Cited by-同舟云学术

Longitudinal evaluation of iron concentration and atrophy in the dentate nuclei in friedreich ataxia

Published:2019-01-09 Issue:3 Volume:34 Page:335-343
ISSN:0885-3185
Container-title:Movement Disorders
language:en
Short-container-title:Mov Disord.

Author:

Ward Phillip G. D.¹²³^ORCID,Harding Ian H.²^ORCID,Close Thomas G.¹,Corben Louise A.²⁴⁵,Delatycki Martin B.²⁴⁵⁶,Storey Elsdon⁷,Georgiou‐Karistianis Nellie²,Egan Gary F.¹²³

Affiliation:

1. Monash Biomedical Imaging, Monash University Melbourne Australia

2. School of Psychological Sciences and Monash Institute of Cognitive and Clinical Neurosciences, Monash University Melbourne Australia

3. Australian Research Council Centre of Excellence for Integrative Brain Function Melbourne Australia

4. Bruce Lefroy Centre for Genetic Health ResearchMurdoch Childrens Research Institute Melbourne Australia

5. Department of PaediatricsUniversity of Melbourne Parkville Australia

6. Victorian Clinical Genetics Service Parkville Australia

7. Department of MedicineMonash University Melbourne Australia

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/mds.27606

Reference56 articles.

1. Neuroimaging in Ataxias

2. Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion

3. Friedreich Ataxia: Neuropathology Revised

4. Regulation of Mitochondrial Iron Accumulation by Yfh1p, a Putative Homolog of Frataxin

5. Deletion of the yeast homologue of the human gene associated with Friedreich's ataxia elicits iron accumulation in mitochondria

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