Calpain activation in apoptosis of ventral spinal cord 4.1 (VSC4.1) motoneurons exposed to glutamate: Calpain inhibition provides functional neuroprotection-Reference-Cited by-同舟云学术

Calpain activation in apoptosis of ventral spinal cord 4.1 (VSC4.1) motoneurons exposed to glutamate: Calpain inhibition provides functional neuroprotection

Published:2005-08-15 Issue:4 Volume:81 Page:551-562
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Das Arabinda,Sribnick Eric A.,Wingrave James M.,Del Re Angelo M.,Woodward John J.,Appel Stanley H.,Banik Naren L.,Ray Swapan K.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference59 articles.

1. Calpain I activation in rat hippocampal neurons in culture is NMDA receptor selective and not essential for excitotoxic cell death

2. Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function

3. Glutamate neurotoxicity, oxidative stress and mitochondria

4. Calpain inhibitor AK295 protects neurons from focal brain ischemia. Effects of postocclusion intra-arterial administration.

5. Cytochrome c Release and Caspase Activation in Traumatic Axonal Injury

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