Pathophysiological roles of chronic low‐grade inflammation mediators in polycystic ovary syndrome

Author:

Rostamtabar Maryam1,Esmaeilzadeh Sedigheh2,Tourani Mehdi3ORCID,Rahmani Abolfazl1,Baee Masoud1ORCID,Shirafkan Fatemeh3,Saleki Kiarash1,Mirzababayi Sajedeh S.4,Ebrahimpour Soheil5,Nouri Hamid Reza236ORCID

Affiliation:

1. Student Research Committee, Babol University of Medical Sciences Babol Iran

2. Infertility and Reproductive Health Research Center, Health Research Institute Babol University of Medical Science Babol Iran

3. Cellular and Molecular Biology Research Center, Health Research Institute Babol University of Medical Sciences Babol Iran

4. Department of Midwifery Babol University of Medical Sciences Babol Iran

5. Infectious Diseases and Tropical Medicine Research Center, Health Research Institute Babol University of Medical Sciences Babol Iran

6. Immunoregulation Research Center, Health Research Institute Babol University of Medical Sciences Babol Iran

Funder

Babol University of Medical Sciences

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference152 articles.

1. Redox Regulation of NLRP3 Inflammasomes: ROS as Trigger or Effector?

2. Serum and follicular fluid cytokines in polycystic ovary syndrome during stimulated cycles;Amato G.;Obstetrics and Gynecology,2003

3. Antioxidants and management of polycystic ovary syndrome in Iran: A systematic review of clinical trials;Amini L.;Iranian Journal of Reproductive Medicine,2015

4. The prevalence of polycystic ovary syndrome (PCOS) in high school students in Rasht in 2009 according to NIH criteria;Asgharnia M.;International Journal of Fertility & Sterility,2011

5. The Prevalence and Features of the Polycystic Ovary Syndrome in an Unselected Population

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