Failure to detect amphetamine-induced dopamine release in the cortex with [11 C]FLB 457 positron emission tomography (PET): Methodological considerations
Author:
Affiliation:
1. Department of Radiology; University of Pittsburgh; Pittsburgh Pennsylvania
2. Department of Psychiatry; NYU Langone Medical Center; New York New York
3. Department of Psychiatry; University of Pittsburgh; Pittsburgh Pennsylvania
Funder
National Institute on Drug Abuse
National Institute of Mental Health
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/syn.22037/fullpdf
Reference28 articles.
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2. Reproducibility of striatal and thalamic dopamine D2 receptor binding using [11C]raclopride with high-resolution positron emission tomography;Alakurtti;Journal of Cerebral Blood Flow & Metabolism,2011
3. Long-term test-retest reliability of striatal and extrastriatal dopamine D2/3 receptor binding: Study with [(11)C]raclopride and high-resolution PET;Alakurtti;Journal of Cerebral Blood Flow & Metabolism,2015
4. Early pharmacokinetics and clinical effects of oral D-amphetamine in normal subjects;Angrist;Biological Psychiatry,1987
5. Oral D-amphetamine causes prolonged displacement of [11C]raclopride as measured by PET;Cardenas;Synapse (New York, N.Y.),2004
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1. Amphetamine-induced dopamine release in rat: Whole-brain spatiotemporal analysis with [11C]raclopride and positron emission tomography;Journal of Cerebral Blood Flow & Metabolism;2023-10-26
2. Prefrontal and Striatal Dopamine Release Are Inversely Correlated in Schizophrenia;Biological Psychiatry;2022-11
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4. Distinguishing Schizophrenia Subtypes: Can Dopamine Imaging Improve the Signal-to-Noise Ratio?;Biological Psychiatry;2020-02
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