Metallothionein‐I/II expression associates with the astrocyte DNA damage response and not Alzheimer‐type pathology in the aging brain-Reference-Cited by-同舟云学术

Metallothionein‐I/II expression associates with the astrocyte DNA damage response and not Alzheimer‐type pathology in the aging brain

Published:2018-08-10 Issue:11 Volume:66 Page:2316-2323
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Waller Rachel¹,Murphy Mark¹,Garwood Claire J¹,Jennings Luke¹,Heath Paul R¹,Chambers Annabelle¹,Matthews Fiona E²³,Brayne Carol⁴,Ince Paul G¹,Wharton Stephen B¹,Simpson Julie E¹^ORCID,

Affiliation:

1. Sheffield Institute for Translational NeuroscienceUniversity of Sheffield Sheffield United Kingdom

2. MRC Biostatistics UnitUniversity of Cambridge Cambridge United Kingdom

3. Institute of Health and SocietyUniversity of Newcastle Newcastle United Kingdom

4. Institute of Public HealthUniversity of Cambridge Cambridge United Kingdom

Funder

Medical Research Council

Alzheimer's Society

Nottingham University Hospitals NHS Trust

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.23465

Reference48 articles.

1. Increased Density of Metallothionein I/II-Immunopositive Cortical Glial Cells in the Early Stages of Alzheimer's Disease

2. Extremely low-frequency electromagnetic field induces neural differentiation of hBM-MSCs through regulation of (Zn)-metallothionein-3

3. Metallothionein and a peptide modeled after metallothionein, EmtinB, induce neuronal differentiation and survival through binding to receptors of the low-density lipoprotein receptor family

4. Regulation of metallothionein gene expression by oxidative stress and metal ions

5. Mammalian MT1 and MT2 metallothioneins differ in their metal binding abilities

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