Heterozygous inactivation of Gnas in adipose-derived mesenchymal progenitor cells enhances osteoblast differentiation and promotes heterotopic ossification-Reference-Cited by-同舟云学术

Heterozygous inactivation of Gnas in adipose-derived mesenchymal progenitor cells enhances osteoblast differentiation and promotes heterotopic ossification

Published:2011-10-20 Issue:11 Volume:26 Page:2647-2655
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Pignolo Robert J,Xu Meiqi,Russell Elizabeth,Richardson Alec,Kaplan Josef,Billings Paul C,Kaplan Frederick S,Shore Eileen M

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jbmr.481/fullpdf

Reference35 articles.

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5. Physiological functions of the imprinted Gnas locus and its protein variants Gα(s) and XLα(s) in human and mouse;Plagge;J Endocrinol.,2008

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